Time tested remedies | Most affordable prices

Male Sterility

Condition Overview:

The condition in which a married couple finds it difficult to produce an offspring is called sterility .The defect may be either with the male partner or the female partner. These defects can be either organic or functional .In males the procreative factors is the sperm. It is produced in the testicles and ejaculated through the male genital organ during sexual intercourse. The production of sperm is regulated by hormones which are secreted by the ductless glands of the body .For the procreation of offspring the sperm should be active and they should be in sufficient numbers in the semen. Sometimes because of some morbidity, sperm is either absent in the semen or small in number. In such cases conception does not take place.

As far as the male causes of infertility are concerned, frequent assume an ingravescent character in the time and become decisive because of the current social tendency to procrastinate the search for the right moment for pregnancy.

Therefore, as may happen to the woman, the delay in the wedding age certainly constitutes the principle factor of sterility in the industrialised countries.

In the following table, the principle causes of male infertility accordingly refer to personal case studies (observed in 600 cases of male infertility)

Andrological pathologies in male infertility are shown to be:

(individual or in association with others, %)

• clinical varicocele 19.3% (=varicose veins of the testicles)
• infection of the seminal tracts 14.7% (prostatitis, epididimitis, vescicolitis, urethritis etc)
• testicular hypotrophy without evident causes 11% (reduction in the testicle development)
• testicular retractibility, former or active 8.8% (=the testicles which come back to or up from the abdomen)
• scrotal hypoplasia with a "high" location of the gonads 8.5% (=small testicle placed too close to the abdomen with a consequent augmentation of temperature)
• infraclinical varicocele 7.3% (small varicose veins of the testicle)
• environmental, professional, iatrogenic (that is, drug-related) damage 3.3%
• former criptorchidism of one or both testicles (undescended testes) 2.7% (=testicles that do not descend in the scrotal sac at the moment of birth, which or is cured too late or not at all)
• acquired obstruction of the seminal pathways [tracts] 0.6% (=inflammatory and infectious factors which compromise the perviousness of the seminal pathways impeding the leaking of spermatozoa from the testicles)
• secondary hypogonadism (completed or strained form) 0.9% (=low level of testosterone from endocrinological causes)
• dysgenic causes 0.3% (irreversible, from the chromosomes)
• chriptorchidism 0.6% (=absence of testicle in the scrotal sac due to being retained in the abdomen)
• immunological factors 0.6% (=presence of antibodies against the spermatozoa often after local infections which take place for a long time without diagnosis)

The condition in which the sperm does not exist in the semen is difficult to treat. But if the sperm are present, but they are either few in number, or inactive, the condition can be easily remedied.