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Anemia, Hereditary Hemolytic

Treatment No. : RD0019
Dosage & Instructions: 15 drops in 15 ml of plain water twice a day, mornings and evenings.
Composition:
Camphor D20+200C
Thuja D10+100C
Pulsatilla D10+100C
Kali brom D10+100C
Symphytum D10+100C
Phosphorus D10+100C
Helonias D10+100C
Treatment Type:
This remedy is a symptomatic treatment. A symptomatic treatment cannot eliminate the disease from its root but is able to considerably improve the patient's condition by easing his or her symptoms.
Contra-indications: None established
Special Instructions: Whereas this remedy can be safely used by teenagers, its use has not been evaluated in children under 12 years of age. Please do not administer to children under this age.
Storage: Store in a cool and dry place
Precautions: Keep away from the reach of children
Standard Packaging : 30 ml Drops

Order Online:

One 30 ml bottle for $79.99
Two 30 ml bottles for $129.99

 

Condition Overview:

Hereditary Nonspherocytic Hemolytic Anemia is a term used to describe a group of rare genetic blood disorders characterized by defective red blood cells (erythrocytes) that are not abnormally "sphere-shaped" (spherocytes). These disorders are thought to be caused by defects in the membranes of red blood cells, abnormal metabolism of a chemical contained in hemoglobin (porphyrin), and deficiencies in certain enzymes such as glucose-6-phosphate dehydrogenase (G6PD) or pyruvate kinase. There are approximately 16 red blood cell enzyme abnormalities that may cause Hereditary Nonspherocytic Hemolytic Anemia.

Glucose-6-Phosphate Dehydrogenase Deficiency is one of the most common inherited enzyme abnormalities in humans. There are close to 300 different varieties of enzymatic disorders that have been classified into 5 main groups according to their relationship to red blood cells and the premature destruction of red blood cells (hemolysis). Generally people with Hereditary Nonspherocytic Hemolytic Anemia who also have a glucose-6-phosphate dehydrogenase deficiency have inherited an uncommon variant of this enzyme deficiency characterized by decreased enzyme activity, extremely abnormal kinetics, and reduced heat stability. These factors could account for the defective function of glucose-6-phosphate dehydrogenase.

 

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